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1 Νοε 2015 · This article will explore several common theories about why and how we age. By understanding current aging theories, clinicians can follow advances in aging research and intelligently address patient questions about life extension therapies and strategies to slow aging.
- Biological Theories of Aging
Here, we review known biological theories of aging and how...
- If You Would Live Long, Choose Your Parents Well
The first question is addressed by the evolutionary theories...
- Thymic Involution With Ageing
Role of the thymus in T-ceiE development The thymus plays a...
- Biological Theories of Aging
Modern biological theories of aging in humans fall into two main categories: programmed and damage or error theories. The programmed theories imply that aging follows a biological timetable, perhaps a continuation of the one that regulates childhood growth and development.
20 Ιουλ 2022 · Ageing. Complexity. Emergence. Abstract. Having made substantial progress understanding molecules, cells, genes and pathways, aging biology research is now moving toward integration of these...
This review critically examines the role of aging in the progressive decline of the mechanobiology occurring in cells, and establish mechanistic frameworks to understand the mechanobiological impacts of aging on disease progression and develop new strategies to halt and reverse the aging process.
1 Οκτ 2010 · Abstract. Despite recent advances in molecular biology and genetics, the mysteries that control human lifespan are yet to be unraveled. Many theories, which fall into two main categories: programmed and error theories, have been proposed to explain the process of aging, but neither of them appears to be fully satisfactory.
21 Ιαν 2021 · Here, through the lens of telomere biology, we examine how telomere dysfunction may amplify or drive molecular biological processes underlying each hallmark of aging and contribute to development of age-related diseases such as neurodegeneration and cancer.
1 Νοε 2015 · Here, we review known biological theories of aging and how ROS mechanistically control senescence and the aging process. We also describe the redox-regulated signaling networks controlling the SASP and its important role in driving age-related diseases.
