Αποτελέσματα Αναζήτησης
17 Αυγ 2022 · Cerebral injury (cerebral edema) is an uncommon but potentially devastating consequence of diabetic ketoacidosis (DKA). This complication is far more common among children with DKA than among adults. Children presenting with more severe DKA (higher blood urea nitrogen levels and more severe acidosis and hypocapnia) are at greatest risk [ 1-4 ].
Objective: To review the causes of cerebral edema in diabetic ketoacidosis (CEDKA), including pathophysiology, risk factors, and proposed mechanisms, to review the diagnosis, treatment, and prognosis of CEDKA and the treatment of diabetic ketoacidosis as it pertains to prevention of cerebral edema. Data source: A MEDLINE search using OVID was ...
Fourth, data suggest that the predominant pathophysiology early after initial cerebral injury (broadly defined) is cerebral ischemia, followed in the subacute stages by a more vasogenic process. Finally, impaired cerebral autoregulation is documented in both TBI and DKA, either reflecting the disease severity or playing a role in the ...
1 Φεβ 2000 · The case for a multifactorial cause of cerebral edema in DKA. Mild edema and other histopathology exists before treatment. Rare reports of symptomatic cerebral edema presenting before any possible influence of therapy on the brain. Inconsistent correlations of cerebral edema with serum. Osmolality. Sodium concentration. Acid base status.
Cerebral edema (CE) is a potentially devastating complication of diabetic ketoacidosis (DKA) that almost exclusively occurs in children. Since its first description in 1936, numerous risk factors have been identified; however, there continues to be ...
The excellent evidence based review of the emergency management of diabetic ketoacidosis (DKA) in adults by Hardern and Quinn perpetuates the premise that “unnecessarily large volumes of intravenous fluids should be avoided because of the high case fatality rate of cerebral oedema”. 1 This presupposes that the rate of fluid delivery is ...
21 Αυγ 2023 · Up to 1% of DKA patients will have cerebral edema due to rapid osmolar shifts. Look for signs of sudden increased intracranial pressure: bradycardia, headache, papilledema, irritability, rising blood pressure, and decreasing Glasgow coma scale (GCS).
