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Diabetes insipidus (DI) is characterized by the decreased ability of the kidneys to concentrate urine. Antidiuretic hormone (ADH) is the primary determinant of free water balance. ADH is produced in posterior pituitary and acts on the V2 receptors of the collecting tubules of the kidney.
This document replaces the “Practice Guidelines for Sedation and Analgesia by Non-Anesthesiologists: An Updated Report by the American Society of Anesthesiologists (ASA) Task Force on Sedation and Analgesia by Non-Anesthesiologists,” adopted in 2001 and published in 2002. 1. Methodology. Definition of Procedural Moderate Sedation and Analgesia.
1 Ιουν 2023 · Diabetes insipidus related to anesthetics and sedatives is an emerging topic in the anesthesia literature. •. Sedatives have been suspected of causing diabetes insipidus in 21 patients in 17 case reports in the ICU literature. •. Dexmedetomidine and sevoflurane were implicated as the causative agent in the majority of case reports. •.
Results: We identified 21 cases of diabetes insipidus or polyuria in the ICU attributed to a sedative. Dexmedetomidine was implicated in 42.9% of cases, followed by sevoflurane (33.3%) and ketamine (23.8%).
Practice has changed in ICUs with the goal to minimise sedation in order to reduce length of stay (LOS) and delirium. 1 This should result in decreased benzodiazepine use, 2 and volatile anaesthetic agents could provide a safe alternative.
2 Φεβ 2023 · To identify cases of diabetes insipidus (DI) related to sedation in the ICU to determine which medications pose the greatest risk and understand patterns of presentation. Materials and methods.
13 Οκτ 1999 · The qualified practitioner corrects adverse physiologic consequences of the deeper-than-intended level of sedation (such as hypoventilation, hypoxia and hypotension) and returns the patient to the originally intended level of sedation.
