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1 Φεβ 2024 · GERD pathophysiology is complex and multifactorial. Major players are the transdiaphragmatic pressure gradient and the valve mechanism at the EGJ. GERD may occur in specific clinical scenarios: obesity, restrictive pulmonary diseases, hiatal hernia, and esophageal and gastric dysmotility.
6 Ιουν 2020 · The pathophysiology of GERD is multifactorial and is best explained by various mechanisms involved, including the influence of the tone of the lower esophageal sphincter, the presence of a hiatal hernia, esophageal mucosal defense against the refluxate and esophageal motility.
Gastroesophageal reflux disease (GERD) is a specific clinical entity defined by the occurrence of gastro-esophageal reflux through the lower esophageal sphincter (LES) into the esophagus or oropharynx to cause symptoms, injury to esophageal tissue, or both.The pathophysiology of GERD is complex and not completely understood.An abnormal LES pres-
The pathogenesis of gastroesophageal reflux disease (GERD) is complex and involves changes in reflux exposure, epithelial resistance, and visceral sensitivity. The gastric refluxate is a noxious material that injures the esophagus and elicits symptoms.
Gastroesophageal Reflux Disease (GERD) can be caused by foods, smoking cigarettes, hypertension, delayed gastric emptying, and abnormal relaxation of the Lower Esophageal Sphincter. Repeated damage to the esophagus from stomach acid causes scarring that can lead to Barrett's Esophagus, a precancerous lesion.
The pathogenesis of gastroesophageal reflux disease (GERD) is complex and involves changes in reflux exposure, epithelial resistance, andvisceral sensitivity.
1 Ιαν 2018 · The pathogenesis of gastroesophageal reflux disease (GERD) is complex and involves changes in reflux exposure, epithelial resistance, and visceral sensitivity. The gastric refluxate is a noxious material that injures the esophagus and elicits symptoms.
