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Basic principles. HR should be corrected slowly (particularly if HR is of unknown duration or chronic) as rapid correction can induce cerebral edema, seizures, permanent neurological damage and death (rate of correction of Na should be <0.5 mmol/l/hour or <12 mmol/l/day).
17 Μαΐ 2024 · This topic will focus on the treatment of hypernatremia induced by water loss, which is the most common cause. The treatment of hypernatremia in patients with impaired thirst, with or without arginine vasopressin disorders, and with primary sodium overload will also be reviewed.
1 Φεβ 2012 · Chronic hypernatremia can be treated safely and effectively using sound clinical reasoning and quantitative assessment of intake and output, particularly urinary output. All forms of hypernatremia are associated with a relative or absolute deficiency of water.
The review presents the main pathogenetic mechanisms of hypernatremia, provides specific directions for the evaluation of patients with increased sodium levels and describes a detailed algorithm for the proper correction of hypernatremia.
We summarize the eight diagnostic steps of the traditional approach and introduce new biomarkers: exclude pseudohypernatremia, confirm glucose-corrected sodium concentrations, determine the extracellular volume status, measure urine sodium levels, measure urine volume and osmolality, check ongoing urinary electrolyte free water clearance, determ...
Enteral and parenteral nutrition regimens can be modified to help restore normal plasma sodium concentrations. Response to treatment requires close monitoring to avoid potential complications associated with rapid correction of hyponatremia and hypernatremia.
10 Οκτ 2019 · Overall body fluid concentration is regulated within a narrow range by the concerted action of the hypothalamic-pituitary axis to influence water intake through thirst and water excretion via the effect of vasopressin, or antidiuretic hormone, on renal collecting duct water permeability.
