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Someone with Mitochondrial myopathy who is generally on a high protein, low sugar diet finds that when she needs an IV, switching to dextrose (sugar) and water rather than saline makes her feel better - why is this?
Resistance training in Mitochondrial myopathy • Group of 8 patients:(39+9 y) with single large-scale deletions. • Training Protocol – Bilateral leg extension/flexion, leg press – 12 weeks, 3 x per week at 80-85% 1RM (3 to 6 sets, 6-8 reps) Double-leg press 1 RM (pounds)
Certain dietary supplements may be of value in treating PMDs because, as nutrients and metabolic cofactors, they help increase mitochondrial ATP production, bypass a cellular defect (e.g., a deficiency in the activity of complexes I, II, or III in the ETC), or remove toxic metabolites [9, 14].
12 Ιουν 2024 · Mitochondrial disease results from failure of mitochondria to function properly. This can lead to less energy, cell injury and cell death. The most common organs that may experience damage are the brain, heart, liver, muscles, kidneys and the endocrine system.
It is especially important to fully understand the safety and efficacy of dietary supplements and medical foods used in the care of mitochondrial patients to better inform decisions made by patients and clinicians about the best use of these management modalities.
Diet: Dietary needs are extremely variable from patient to patient with mitochondrial myopathies. It is required to evaluate a patient’s nutritional deficiencies. Potential therapies include supplementing caloric intake, enteral feeding, increasing the frequency of meals, limited fasting, and parenteral nutrition ( Parikh et al., 2009 ).
The estimate of nutritional status, en-ergy needs and nutritional behaviors are first-ly discussed. Then, we go in-depth on the sci-entific rationale and the clinical evidence of the use of anti-oxidants and enzyme-cofactors in the clinical practice.
