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Fourth, data suggest that the predominant pathophysiology early after initial cerebral injury (broadly defined) is cerebral ischemia, followed in the subacute stages by a more vasogenic process.
- Brain injury in children with diabetic ketoacidosis: Review of the ...
This review critically examines the literature on the...
- Cerebral edema in diabetic ketoacidosis - PubMed
Objective: To review the causes of cerebral edema in...
- Brain injury in children with diabetic ketoacidosis: Review of the ...
17 Αυγ 2022 · Subclinical cerebral edema, as detected by ventricular narrowing on imaging studies or alterations in brain water distribution on magnetic resonance imaging (MRI), has been reported in the majority of children with DKA, even in the absence of neurologic signs or symptoms .
1 Φεβ 2000 · The case for a multifactorial cause of cerebral edema in DKA. Mild edema and other histopathology exists before treatment. Rare reports of symptomatic cerebral edema presenting before any possible influence of therapy on the brain. Inconsistent correlations of cerebral edema with serum. Osmolality. Sodium concentration. Acid base status.
This review critically examines the literature on the pathophysiology of CE and attempts to categorize the findings by types of brain injury that contribute to its development: cytotoxic, vasogenic, and osmotic. Utilizing this scheme, we propose a multifactorial pathway for the development of CE in patients with DKA.
18 Δεκ 2018 · Treatment of cerebral edema in DKA with hyperosmolar agents is recommended as the pathophysiology of cerebral edema is not very well understood.6, 16, 17, 18, 19 An ischemic6 and/or vasogenic18 process is proposed to play a role in the genesis of DKA related cerebral edema.
Objective: To review the causes of cerebral edema in diabetic ketoacidosis (CEDKA), including pathophysiology, risk factors, and proposed mechanisms, to review the diagnosis, treatment, and prognosis of CEDKA and the treatment of diabetic ketoacidosis as it pertains to prevention of cerebral edema. Data source: A MEDLINE search using OVID was ...
In DKA, cerebral edema usually occurs early (occurring within the first 7-8 hours in approximately 2/3rd) in the treatment of DKA with the remaining cases occurring up to 28-30 hours after fluid resuscitation and initiation of insulin treatment [3,6].
