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Fourth, data suggest that the predominant pathophysiology early after initial cerebral injury (broadly defined) is cerebral ischemia, followed in the subacute stages by a more vasogenic process.
- Cerebral edema in diabetic ketoacidosis - PubMed
Objective: To review the causes of cerebral edema in...
- Brain injury in children with diabetic ketoacidosis: Review of the ...
This review critically examines the literature on the...
- How can cerebral edema during treatment of diabetic ... - PubMed
This article will discuss how DKA might be prevented from...
- Cerebral edema in diabetic ketoacidosis - PubMed
17 Αυγ 2022 · Symptoms of cerebral injury may be present prior to initiation of therapy or emerge during treatment. In most cases, symptoms become apparent within 12 hours after the initiation of therapy and, rarely, later than 24 hours . The pathophysiology, diagnosis, and treatment of cerebral injury in children with DKA will be discussed here.
Objective: To review the causes of cerebral edema in diabetic ketoacidosis (CEDKA), including pathophysiology, risk factors, and proposed mechanisms, to review the diagnosis, treatment, and prognosis of CEDKA and the treatment of diabetic ketoacidosis as it pertains to prevention of cerebral edema. Data source: A MEDLINE search using OVID was ...
This review critically examines the literature on the pathophysiology of CE and attempts to categorize the findings by types of brain injury that contribute to its development: cytotoxic, vasogenic, and osmotic. Utilizing this scheme, we propose a multifactorial pathway for the development of CE in patients with DKA.
1 Φεβ 2000 · Treatment guidelines for DKA include slow rehydration with the goal of gradually decreasing serum osmolality. In the absence of a good understanding of the pathogenesis of cerebral edema in DKA, this recommendation is prudent. In my own practice, I aim to rehydrate such patients over 36–48 h.
This article will discuss how DKA might be prevented from occurring in the first instance, known risk factors for cerebral edema, fluid and insulin management, the importance of careful monitoring during DKA treatment, and the importance of recognizing and acting on the earliest symptoms to prevent long-term harm.
The essential components of the treatment regimen for DKA are fluid and electrolyte resuscitation and insulin therapy. Cerebral edema manifests clinically in approximately 1% of DKA patients with progression to brain herniation, representing the major cause of mortality and morbidity in DKA [4].
