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  1. 17 Μαΐ 2024 · This topic will focus on the treatment of hypernatremia induced by water loss, which is the most common cause. The treatment of hypernatremia in patients with impaired thirst, with or without arginine vasopressin disorders, and with primary sodium overload will also be reviewed.

  2. 24 Αυγ 2023 · The goal of therapy is to correct both the serum sodium and the intravascular volume. Fluids should be administered orally or via a feeding tube whenever possible. In patients with severe dehydration or shock, the initial step is fluid resuscitation with isotonic fluids before free water correction.

  3. The cerebral adaptation and clinical manifestations of hyponatremia and hypernatremia will be reviewed here. The etiology and treatment of hyponatremia and hypernatremia are presented elsewhere: (See "Causes of hypotonic hyponatremia in adults".) (See "Overview of the treatment of hyponatremia in adults".)

  4. 25 Οκτ 2023 · Treatment of hypernatraemia is directed at addressing the underlying cause, as well as replacing free water deficit and ongoing losses while monitoring serum sodium concentration. It is important not to correct the serum sodium concentration too rapidly in cases of chronic hypernatraemia.

  5. sso.uptodate.com › contents › general-principles-of-disorders-of-water-balanceUpToDate

    8 Οκτ 2024 · Understanding these basic principles is essential for appropriate diagnosis and treatment. The general principles and disorders of water balance and sodium balance will be reviewed here. The causes and evaluation of hyponatremia, hypernatremia, hypovolemia, and edema are presented separately:

  6. The review presents the main pathogenetic mechanisms of hypernatremia, provides specific directions for the evaluation of patients with increased sodium levels and describes a detailed algorithm for the proper correction of hypernatremia.

  7. This topic will focus on the treatment of hypernatremia induced by water loss, which is the most common cause. The treatment of hypernatremia in patients with impaired thirst, with or without diabetes insipidus, and with primary sodium overload will also be reviewed.

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