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In chemistry, cyanide (from Greek kyanos 'dark blue') is a chemical compound that contains a C≡N functional group. This group, known as the cyano group, consists of a carbon atom triple-bonded to a nitrogen atom. [1] In inorganic cyanides, the cyanide group is present as the cyanide anion − C≡N. This anion is extremely poisonous.
17 Σεπ 2018 · Cyanide can refer to any chemical that contains a carbon-nitrogen (CN) bond. Here’s how to identify the symptoms of poisoning, who’s at risk, and more. Healthline
Cyanide poisoning is poisoning that results from exposure to any of a number of forms of cyanide. [4] Early symptoms include headache, dizziness, fast heart rate, shortness of breath, and vomiting. [2] This phase may then be followed by seizures, slow heart rate, low blood pressure, loss of consciousness, and cardiac arrest. [2]
24 Ιουλ 2024 · Many substances contain cyanide, but not all of them are deadly poisons. Sodium cyanide (NaCN), potassium cyanide (KCN), hydrogen cyanide (HCN), and cyanogen chloride (CNCl) are lethal, but thousands of compounds called nitriles contain the cyanide group yet aren't as toxic.
13 Φεβ 2023 · Cyanide is a rapidly acting substance that is traditionally known as a poison. Hydrogen cyanide was first isolated from Prussian blue dye in 1786, and cyanide first extracted from almonds around 1800. Cyanide can exist as a gas, hydrogen cyanide, a salt, potassium cyanide.
12 Νοε 2024 · cyanide, any compound containing the monovalent combining group CN. In inorganic cyanides, such as sodium cyanide (NaCN), this group is present as the negatively charged cyanide ion; these compounds, which are regarded as salts of hydrocyanic acid, are highly toxic.
6 Σεπ 2024 · Cyanide is a fast acting and potentially deadly chemical that affects the body's ability to use oxygen. It comes from natural substances in some foods and in certain plants, including the pits and seeds of some common fruits. Cyanide is sometimes described as having a "bitter almond" smell.
Cyanide is a very poisonous chemical. Exposure to high levels of cyanide harms the brain and heart, and may cause coma and death. Exposure to lower levels may result in breathing difficulties, heart pains, vomiting, blood changes, headaches, and enlargement of the thyroid gland.
Management of cyanide toxicity is based on the principle of reversing and/or displacing cyanide binding to cytochrome a3. There are two major modalities of treatment: hydroxocobalamin and
Cyanide is an irreversible inhibitor of the enzyme cytochrome c oxidase (also known as aa 3) in the fourth complex in the membrane of the mitochondria of cells. It attaches to the iron within this protein. The binding of cyanide to this cytochrome prevents transport of electrons from cytochrome c oxidase to oxygen.
